Chronological brain lesions after SARS-CoV-2 infection in hACE2-transgenic mice


Por: Vidal, E, Lopez-Figueroa, C, Rodon, J, Perez, M, Brustolin, M, Cantero, G, Guallar, V, Izquierdo-Useros, N, Carrillo, J, Blanco, J, Clotet, B, Vergara-Alert, J and Segales, J

Publicada: 1 jul 2022 Ahead of Print: 1 dic 2021
Resumen:
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) causes respiratory disease, but it can also affect other organs including the central nervous system. Several animal models have been developed to address different key questions related to Coronavirus Disease 2019 (COVID-19). Wild-type mice are minimally susceptible to certain SARS-CoV-2 lineages (beta and gamma variants), whereas hACE2-transgenic mice succumb to SARS-CoV-2 and develop a fatal neurological disease. In this article, we aimed to chronologically characterize SARS-CoV-2 neuroinvasion and neuropathology. Necropsies were performed at different time points, and the brain and olfactory mucosa were processed for histopathological analysis. SARS-CoV-2 virological assays including immunohistochemistry were performed along with a panel of antibodies to assess neuroinflammation. At 6 to 7 days post inoculation (dpi), brain lesions were characterized by nonsuppurative meningoencephalitis and diffuse astrogliosis and microgliosis. Vasculitis and thrombosis were also present and associated with occasional microhemorrhages and spongiosis. Moreover, there was vacuolar degeneration of virus-infected neurons. At 2 dpi, SARS-CoV-2 immunolabeling was only found in the olfactory mucosa, but at 4 dpi intraneuronal virus immunolabeling had already reached most of the brain areas. Maximal distribution of the virus was observed throughout the brain at 6 to 7 dpi except for the cerebellum, which was mostly spared. Our results suggest an early entry of the virus through the olfactory mucosa and a rapid interneuronal spread of the virus leading to acute encephalitis and neuronal damage in this mouse model.

Filiaciones:
Vidal, E:
 UAB, Ctr Recerca Sanitat Anim CReSA, IRTA, Campus UAB, Barcelona 08193, Catalonia, Spain

Lopez-Figueroa, C:
 UAB, Ctr Recerca Sanitat Anim CReSA, IRTA, Campus UAB, Barcelona 08193, Catalonia, Spain

Rodon, J:
 UAB, Ctr Recerca Sanitat Anim CReSA, IRTA, Campus UAB, Barcelona 08193, Catalonia, Spain

Perez, M:
 UAB, Ctr Recerca Sanitat Anim CReSA, IRTA, Campus UAB, Barcelona 08193, Catalonia, Spain

Brustolin, M:
 UAB, Ctr Recerca Sanitat Anim CReSA, IRTA, Campus UAB, Barcelona 08193, Catalonia, Spain

Cantero, G:
 UAB, Ctr Recerca Sanitat Anim CReSA, IRTA, Campus UAB, Barcelona 08193, Catalonia, Spain

Guallar, V:
 Barcelona Supercomp Ctr BSC, Barcelona, Spain

 Catalan Inst Res & Adv Studies ICREA, Barcelona, Spain

:
 irsiCaixa AIDS Res Inst, Badalona, Spain

 Germans Trias & Pujol Res Inst IGTP, Can Ruti Campus, Badalona, Spain

:
 irsiCaixa AIDS Res Inst, Badalona, Spain

:
 irsiCaixa AIDS Res Inst, Badalona, Spain

 Germans Trias & Pujol Res Inst IGTP, Can Ruti Campus, Badalona, Spain

 Univ Vic Cent Univ Catalonia UVic UCC, Vic, Spain

:
 irsiCaixa AIDS Res Inst, Badalona, Spain

 Germans Trias & Pujol Res Inst IGTP, Can Ruti Campus, Badalona, Spain

 Univ Vic Cent Univ Catalonia UVic UCC, Vic, Spain

Vergara-Alert, J:
 UAB, Ctr Recerca Sanitat Anim CReSA, IRTA, Campus UAB, Barcelona 08193, Catalonia, Spain

Segales, J:
 UAB, Ctr Recerca Sanitat Anim CReSA, IRTA, Campus UAB, Barcelona 08193, Catalonia, Spain

 UAB, Dept Sanitat & Anat Anim, Fac Vet, Campus UAB, Barcelona, Catalonia, Spain
ISSN: 03009858





Veterinary Pathology
Editorial
SAGE Publications, 2455 TELLER RD, THOUSAND OAKS, CA 91320 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 59 Número: 4
Páginas: 613-626
WOS Id: 000738352400001
ID de PubMed: 34955064
imagen Green Published, hybrid

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