Comparison of the Effectiveness and Safety of the Oral Selective Inhibitor of Nuclear Export, Selinexor, in Diffuse Large B Cell Lymphoma Subtypes.


Por: Casasnovas RO, Follows G, Zijlstra JM, Vermaat JSP, Kalakonda N, Choquet S, Neste EVD, Hill B, Thieblemont C, Cavallo F, la Cruz F, Kuruvilla J, Hamad N, Jaeger U, Caimi PF, Gurion R, Warzocha K, Bakhshi S, Sancho JM, Schuster M, Egyed M, Offner F, Vassilakopoulos TP, Samal P, Ku M, Ma X, Chamoun K, Shah J, Canales M, Maerevoet M, Shacham S, Kauffman MG and Goy A

Publicada: 1 ene 2022 Ahead of Print: 22 jul 2021
Resumen:
BACKGROUND: The SADAL study evaluated oral selinexor in patients with relapsed and/or refractory diffuse large B-cell lymphoma (DLBCL) after at least 2 prior lines of systemic therapy. In this post-hoc analysis, we analyzed the outcomes of the SADAL study by DLBCL subtype to determine the effects of DLBCL subtypes on efficacy and tolerability of selinexor. PATIENTS AND METHODS: Data from 134 patients in SADAL were analyzed by DLBCL subtypes for overall response rate (ORR), overall survival (OS), duration of treatment response, progression-free survival, and adverse events rate. RESULTS: ORR in the entire cohort was 29.1%, and similar in patients with germinal center (GCB) versus non-GCB DLBCL (31.7% vs. 24.2%, P = 0.45); transformed DLBCL showed a trend towards higher ORR than de novo DLBCL: 38.7% vs. 26.2% (P = 0.23). Despite similar prior treatment regimens and baseline characteristics, patients with DLBCL and normal C-MYC/BCL-2 protein expression levels had a significantly higher ORR (46.2% vs.14.8%, P = 0.012) and significantly longer OS (medians 13.7 vs. 5.1 months, hazard ratio 0.43 [95% CI, 0.23-0.77], P = 0.004) as compared with those whose DLBCL had C-MYC and BCL-2 overexpression. Among patients who had normal expression levels of either C-MYC or BCL-2 and baseline hemoglobin levels = 10g/dL, ORR was 51.5% (n = 47), with median OS of 15.5 months and median PFS of 4.6 months. Similar rates of adverse events were noted in all subgroups. CONCLUSIONS: Overall, single agent oral selinexor showed strong responses in patients with limited treatment alternatives regardless of germinal center B-cell type or disease origin.

Filiaciones:
Casasnovas RO:
 Hématologie Clinique and INSERM 1231, CHU Dijon Bourgogne, Dijon, France

Follows G:
 Addenbrooke's Hospital, Cambridge, United Kingdom

Zijlstra JM:
 Amsterdam University Medical Center, Vrije Universiteit, Cancer Center, Amsterdam, The Netherlands

Vermaat JSP:
 Leiden University Medical Center, Leiden, The Netherlands

Kalakonda N:
 University of Liverpool, Liverpool, United Kingdom

Choquet S:
 Hôpital Pitié Salpêtrière, Paris, France

Neste EVD:
 Cliniques Universitaires Saint-Luc, Brussels, Belgium

Hill B:
 Cleveland Clinic, Cleveland, OH

Thieblemont C:
 AP-HP, Hopital Saint-Louis, Hémato-oncology, DMU DHI, Paris, France

 Université de Paris, Paris, France

Cavallo F:
 Department of Molecular Biotechnologies and Health Sciences, Division of Hematology, University of Turin, Turin, Italy

la Cruz F:
 Hospital Universitario Virgen del Rocio, Sevilla, Spain

Kuruvilla J:
 Princess Margaret Cancer Center, Toronto, Canada

Hamad N:
 St Vincent's Hospital Sydney, Darlinghurst, NSW, Australia

Jaeger U:
 Medical University of Vienna, Vienna, Austria

Caimi PF:
 UH Seidman Cancer Center, Cleveland, OH

Gurion R:
 Institute of Hematology, Rabin Medical Center, Petah Tikva, Israel

 Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel

Warzocha K:
 Instytut Hematologii i Transfuzjologii, Warsaw, Poland

Bakhshi S:
 Dr. B. R. A. Institute Rotary Cancer Hospital, New Delhi, India

:
 Hospital Universitari Germans Trias i Pujol, Barcelona, Spain

Schuster M:
 Stony Brook University Hospital Cancer Center, New York, NY

Egyed M:
 Teaching Hospital Kaposi Mór, Kaposvár, Hungary

Offner F:
 Ghent University Hospital, Ghent, Belgium

Vassilakopoulos TP:
 Department of Haematology, Laikon General Hospital, National and Kapodistrian University of Athens, Athens, Greece

Samal P:
 Institute of Medical Sciences & SUM Hospital, Odisha, India

Ku M:
 St.Vincent's Hospital Melbourne, Fitzroy, Victoria, Australia

Ma X:
 Karyopharm Therapeutics, Newton, MA

Chamoun K:
 Karyopharm Therapeutics, Newton, MA

Shah J:
 Karyopharm Therapeutics, Newton, MA

Canales M:
 Hospital Universitario La Paz, Madrid, Spain

Maerevoet M:
 Institute Jules Bordet, Brussels, Belgium

Shacham S:
 Karyopharm Therapeutics, Newton, MA

Kauffman MG:
 Karyopharm Therapeutics, Newton, MA

Goy A:
 Hackensack University Medical Center, Hackensack, NJ
ISSN: 21522650





Clinical Lymphoma Myeloma & Leukemia
Editorial
Cancer Media Group, 3500 MAPLE AVENUE, STE 750, DALLAS, TX 75219-3931 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 22 Número: 1
Páginas: 24-33
WOS Id: 000745004100011
ID de PubMed: 34493477
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