Gefitinib or Erlotinib vs Chemotherapy for EGFR Mutation-Positive Lung Cancer: Individual Patient Data Meta-Analysis of Overall Survival


Por: Lee, CK, Davies, L, Wu, YL, Mitsudomi, T, Inoue, A, Rosell, R, Zhou, C, Nakagawa, K, Thongprasert, S, Fukuoka, M, Lord, S, Marschner, I, Tu, YK, Gralla, RJ, Gebski, V, Mok, T and Yang, JCH

Publicada: 1 jun 2017
Resumen:
Background: We performed an individual patient data meta-analysis to examine the impact of first-generation epidermal growth factor receptor (EGFR) tyrosine kinase inhibitor (TKI) therapy on overall survival (OS) in advanced non-small cell lung cancer (NSCLC). Methods: Data from trials comparing EGFR-TKI against chemotherapy in exon 19 deletion (del19) or exon 21 L858R (L858R) EGFR mutations patients were used. We performed Cox regression to obtain hazard ratios (HRs) and 95% confidence intervals (CIs). Impact of postprogression therapies was examined in exploratory analyses. All statistical tests were two-sided. Results: Six eligible trials (gefitinib = 3, erlotinib = 3) included 1231 patients; 632 received EGFR-TKI and 599 received chemotherapy. At a median 35.0 months follow-up, there were 780 deaths and 1004 progressions. There was no difference in OS between EGFR-TKI and chemotherapy (HR = 1.01, 95% CI = 0.88 to 1.17, P = .84). There was also no difference in OS for Del19 (n = 682, HR = 0.96, 95% CI = 0.79 to 1.16, P = .68) and L858R (n = 540, HR = 1.06, 95% CI = 0.86 to 1.32, P = .59) subgroups (Pinteraction = .47), or according to smoking status, sex, performance status, age, ethnicity, or histology. However, EGFR-TKI statistically significantly prolonged progression-free survival (PFS) overall (HR = 0.37, 95% CI = 0.32 to 0.42, P < .001) and in all subgroups. Following progression, 73.8% from the chemotherapy arm received EGFR-TKI, and 65.9% from the EGFR-TKI arm received chemotherapy. Nine percent from the EGFR-TKI arm received no further treatment vs 0.6% from the chemotherapy arm. Following disease progression, patients randomly assigned to EGFR-TKI had shorter OS than those randomly assigned to chemotherapy (12.8 months, 95% CI = 11.4 to 14.3, vs 19.8 months, 95% CI = 17.6 to 21.7). Conclusions: Despite statistically significant PFS benefit, there is no relative OS advantage with frontline gefitinib or erlotinib vs chemotherapy in EGFR-mutated NSCLC. This finding is likely due to the high rate of crossover at progression.

Filiaciones:
Lee, CK:
 Univ Sydney, NHMRC Clin Trials Ctr, Sydney, NSW, Australia

 St George Hosp, Canc Care Ctr, Sydney, NSW, Australia

Davies, L:
 Univ Sydney, NHMRC Clin Trials Ctr, Sydney, NSW, Australia

Wu, YL:
 Guangdong Gen Hosp, Guangdong Lung Canc Inst, Guangzhou, Guangdong, Peoples R China

 Guangdong Acad Med Sci, Guangzhou, Guangdong, Peoples R China

Mitsudomi, T:
 Kinki Univ, Fac Med, Dept Surg, Div Thorac Surg, Sayama, Osaka, Japan

Inoue, A:
 Tohoku Univ, Sch Med, Dept Palliat Med, Sendai, Miyagi, Japan

:
 Germans Trias & Pujol Hlth Sci Syst & Hosp, Catalan Inst Oncol, Barcelona, Spain

Zhou, C:
 Tongji Univ, Shanghai Pulm Hosp, Sch Med, Dept Oncol, Shanghai, Peoples R China

Nakagawa, K:
 Kinki Univ, Fac Med, Dept Med Oncol, Sayama, Osaka, Japan

Thongprasert, S:
 Chiang Mai Univ, Fac Med, Chiang Mai, Thailand

Fukuoka, M:
 Kinki Univ, Fac Med, Dept Med Oncol, Sayama, Osaka, Japan

Lord, S:
 Univ Sydney, NHMRC Clin Trials Ctr, Sydney, NSW, Australia

 Univ Norte Dame, Sch Med, Sydney, NSW, Australia

Marschner, I:
 Univ Sydney, NHMRC Clin Trials Ctr, Sydney, NSW, Australia

 Macquarie Univ, Dept Stat, Sydney, NSW, Australia

Tu, YK:
 Natl Taiwan Univ, Coll Publ Hlth, Inst Epidemiol & Prevent Med, Taipei, Taiwan

Gralla, RJ:
 Jacobi Med Ctr, Albert Einstein Coll Med, New York, NY USA

Gebski, V:
 Univ Sydney, NHMRC Clin Trials Ctr, Sydney, NSW, Australia

Mok, T:
 Chinese Univ Hong Kong, Hong Kong Canc Inst, Dept Clin Oncol, Shatin, Hong Kong, Peoples R China
ISSN: 00278874





JNCI-JOURNAL OF THE NATIONAL CANCER INSTITUTE
Editorial
Oxford University Press, JOURNALS DEPT, 2001 EVANS RD, CARY, NC 27513 USA, Reino Unido
Tipo de documento: Article
Volumen: 109 Número: 6
Páginas:
WOS Id: 000404050300009
ID de PubMed: 28376144
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